Will I Be Next?

by Julia Hanf

The origin of diabetes is somewhat of a mystery. Findings that date back to the 19th century have given science what knowledge it does have about the disease. Diabetes was found to be linked to the pancreas in the early 1900’s by Canadians Frederick Banting and Charles H. Best. It wasn’t until the 1976 that a clarification of causation was expressed by Alexander Beam. In a paper on diabetes, Beam wrote that the susceptibility to develop diabetes was inherited, but that environmental factors could contribute to the onset of the disease.

Type II diabetes is created by increased aging, obesity, poor nutrition, high stress and physical inactivity. All of these conditions can be traced back to one source high acidity. Over-acid lifestyles and food choices have negative impacts on health, which is shown by the rapidly increasing diabetes rates in the country.

What causes diabetes is not clear. Experts can only establish that it has existed since the 19th century. In the early 1900’s, Canadians Frederick Banting and Charles H. Best established the connection between the pancreas and diabetes. It wasn’t until 1976, however, that Alexander Beam wrote a paper referring to diabetes as being an inherited disease. He said that negative environmental factors, like poor nutrition and physical inactivity, were likely to bring on the disease. It’s important to not that not all diabetics are overweight at the time of diagnosis. However, the question remains: does being overweight make you diabetic, or does diabetes make you overweight?

While exact causes of diabetes are not clear, there is speculation concerning obesity and diabetes. It is the classic cyclic question, which comes first. As an individual’s weight increases as result of poor nutrition, over indulgence and an inactive lifestyle, the body responds less and less to its natural metabolic processes. It fails to convert glucose into fuel for the cell. As a result, the glucose is stored as fat, contributing to the existing weight problem. It is acceptable to state that reduction of weight can assist in prevention or management of the disease.

Another speculation regarding the cause of diabetes is raised levels of pH, or acidity in the body. Studies have found that when a person consumes certain foods or drinks with high acidic value, the liver, muscles and fat cells are over-stimulated. The overstimulation signals these cells to release whatever glucose they contain into the bloodstream. This causes high blood sugar levels. The body responds by releasing fat to neutralize the pH levels. When things return to some form of normal, the body stores the fat to protect cells from future blood sugar elevation. As time goes by, the repeating process can lead to other serious health risks, including diabetes. Elevated levels of pH can cause inflammation and damage to the cardiovascular system, endocrine system and circulatory system to name a few.

Furthermore, the role insulin plays in the metabolic process is not fully understood. The common belief is that insulin is needed to regulate blood sugar levels in the body. The term “insulin dependent” was coined in the 1950’s. It gave to the impression that muscle and fat required insulin to unlock the cell, allowing it to convert the glucose. However, current studies conclude that many different cellular bodies transport glucose. Cells need glucose for their cellular respiration process, regardless of insulin presence.

Over stimulation of the bodily tissues through acidic foods can cause a lot of damage, and type II diabetes is just a symptom of an acidic lifestyle. In order to bring the body back into balance, you must include alkalizing green vegetables, green drinks and good fats in your diet. Plant proteins from grains and legumes also help restore the body’s previous homeostasis. The pH miracle diet includes a balanced plan for eating with your body, instead of against it. With the application of the principles of the diet, controlling and preventing diabetes is a simple matter of alkalized eating and living.

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